Surgery

New born with anorectal malformation

 

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Anorectal Malformations

These are characterized by the absence of the anal opening or an abnormally located anal or rectal opening. These are evident at birth and need urgent attention on behalf of the attending physician. These can be associated with other congenital malformations like cardiac anomalies, gastrointestinal anomalies, vertebral anomalies, genitourinary system anomalies and limb anomalies.

Treatment

  • Isolate the baby,
  • Maintain temperature,
  • Insert nasogastric tube to rule out esophageal atresia and decompress stomach,
  • Reassure and counsell parents .
  • Explain prognosis.

Pharmacological

It is required in all the cases that are to be undertaken for surgery in the form of definitive procedure or preliminary colostomy.

  1. Intravenous fluids 10% Dextrose 60 ml/kg per day (first 48 hours) and Isolyte P 100 ml/kg body weight thereafter till required.
  2. Inj. Vitamin K 1 mg IM stat
  3. Inj. Ampicillin 50-100 mg/kg in 4 divided doses for 7 days.
  4. Inj. Metronidazole 7.5 mg/kg per day in 3 divided doses for 3 days.


Surgical treatment (Best carried out by a qualified paediatric surgeon)

Figure 1 & 2 depict exmamination and management of anorectal anomalies in male and female new borns.Transfer newborns to such centers for performing the definitive surgery. However, colostomy can be performed at places where expertise for doing definitive procedure is not available.

Anovestibular fistula can be managed by cut back procedure at a peripheral center as initial procedure. Anal stenosis can be managed by anal dilatation.
If unsure, it is safe to perform a sigmoid colostomy before referring the patient to a tertiary care center.

Parent education

  • Reassure the parents and explain the nature of anomaly and also explain that the child would require multiple surgical procedures to correct the malformation.
  • Teach the parents the care of the colostomy – not leaving it open, cleaning around it with soft cotton cloth soaked in water, not rubbing over the colostomy to prevent bleeding, management of prolapsed colostomy and follow-up schedule.

References

  1. Anorectal Malformations. In: Paediatric Surgery. O’Neill Jr., Rowe MI, Grosfeld JL et al (eds), 5th Edition, 1998, St. Louis.

New born with anorectal malformation


Figure 1. Male

 


Figure 2. Female

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Undescended Testis

Undescended testis is defined as the testis, which cannot be brought to the base of the scrotum without undue tension on the spermatic cord.

This anomaly is often diagnosed early but the treatment is delayed due to misconceptions leading to various complications.

Salient features

  • The testis can be located in the superficial inguinal pouch,inguinal canal or intra-abdominal site. Truly ectopic testis can be present in perineum, femoral region, pubopenile site or contralateral haemiscrotum.
  • Differentiate from retractile testis which is occasionally pulled up due to reflex contraction of cremasteric muscle. The retractile testis is normal in size, can be brought down in to scrotum where it stays for some time and the scrotum is normally developed.
  • Complications of undescended testis include temperature effects on testis, endocrine effects, germ cell alteration, lower fertility, higher incidence of malignancy, increased incidence of torsion, increased chances of trauma and psychological trauma.

Treatment

  1. If the newborn child is seen with unilateral undescended testis, follow up the patient at intervals to see the descent. If testis fails to descend by the age of 12 months, orchiopexy is advised. If seen after first birthday the operation of orchiopexy should be done before the age of two years. The operation entails mobilizing the testis and cord structures and fixing it in the subdartos pouch in the scrotum with unabsorbable sutures.
  2. If the newborn child has bilateral undescended testes with hypospadias, it should be investigated for intersex disorder.
  3. If a child has undescended testis with clinically visible hernia, orchiopexy can be done at an earlier age along with herniotomy.

Patient education

  • The parents should be informed about the anomaly if detected at birth and advised to monitor the descent of testis and to get it operated by the age of 1 year.

References

Undescended Testis, Torsion and Varicocele. In: Paediatric Surgery, O’Neill Jr, Rowe MI, Grosfeld JL et al (eds), 5th Edition, 1998, Mosby, St. Louis.

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Paediatric surgical conditions-Spina Bifida

Spina bifida is a congenital malformation in which there is incomplete closure of the spinal arch at one or more levels. The disorder can be diagnosed in the antenatal period with ultrasound and a decision regarding continuation of pregnancy can be taken in consultation with paediatric surgeon, obstetrician, neurosurgeon and other specialties.

Salient features

  • Spina bifida occulta-defect is seen only on the radiographs.
    Some sort of cutaneous manifestation may point towards the underlying defect.
  • Spina bifida aperta- meningocele, meningomyelocele and syringomyelocele. This category requires immediate decision about the course of treatment after parent counselling.

The lesions have varying degree of associated neurological deficit, musculoskeletal defects. Almost 90% cases have associated hydrocephalus.

Treatment

  1. If the lesion is detected on antenatal ultrasound, parent counseling should be done after investigations for other associated congenital malformations.
  2. If the parents opt for continuation of pregnancy, they should be referred to a center where facilities of paediatric surgeons or neurosurgeon are available.
  3. If a newborn baby is seen with a defect on the back, baby should be taken for immediate surgery after relevant investigation at a center where expert surgical expertise and operating facilities for neonatal surgery are available. At the peripheral center management would include:
  • Isolation of the newborn and prevention of hypothermia.
  • Care of the lesion and back to prevent desiccation and trauma.
  • Nursing in a prone position.
  • Intravenous fluids -10% Dextrose 60 ml/kg/day for the first 48 hours of life.
  • Inj. Ampicillin (500 mg/vial) 100 mg/kg in 4 divided doses.
  • Transfer the baby to a tertiary care center.

Parent education

  • If diagnosed in antenatal period, parents are counselled and investigated for severity of lesion and possible outcome.
  • Abortion may be advised if associated anomalies are noted in the foetus.

References

  1. Management of Spina Bifida, Hydrocephalus, Central Nervous System Infections and Intractable Epilepsy. In: Paediatric Surgery, O’Neill Jr, Rowe MI, Grosfeld JL et al (eds), 5th Edition, 1998, Mosby, St. Louis.

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Haemorrhoids

Haemorrhoids (commonly called piles) are the dilated tortuous veins occurring in relation to the anus. These can be primary or secondary to some other disease like carcinoma of rectum, pregnancy, straining at micturition, or constipation due to any cause. These can be classified into external, internal or mixed (externo-internal) depending on their position in relation to anal orifice.

Salient features

  • Many small sized haemorrhoids are asymptomatic. They present with bright red painless bleeding that can be mild or severe.
    Mucus discharge, prolapse of piles and occasionally pain can also occur.
  • Chronic cases develop anaemia due to continuous blood loss.
  • On the basis of clinical features, haemorrhoids can be graded -
    - First degree- Bleed only, do not prolapse
    - Second degree- Bleeding occurs, descend down on straining but reduce spontaneously
    - Third degree- Piles prolapse during defecation, but stay prolapsed and have to reposed manually
    - Fourth degree- Piles are large and remain permanently prolapsed
  • On examination, there is no external evidence of haemorrhoids in early cases. In advanced cases, haemorrhoids can be seen on straining or are constantly prolapsed.
  • Complications of haemorrhoids include strangulation, thrombosis, ulceration, gangrene, fibrosis, suppuration and pylophlebitis.

Treatment

Asymptomatic haemorrhoids do not need any treatment. Secondary haemorrhoids due to concomitant disease also tend to resolve once the underlying disease is cured.

In asymptomatic or mild degree haemorrhoids
Bowel regulation by the use of laxatives, use of high fiber diet, sitz bath and application of topical ointment containing Xylocaine (2%) to relieve pain, if any.

In case bleeding persists despite these measures, second and early third degree piles:
Injection treatment using 5% phenol in almond oil (3-5 ml for each pile)/rubber band ligation/photocoagulation/cryosurgery/infrared or laser coagulation.

Late third and fourth degree piles
Haemorrhoidectomy or excision of the piles. The complications of surgery include pain, acute retention of urine, reactive bleeding and later on secondary haemorrhage and anal stricture.

Patient education

  • Avoid constipation and use laxative if required.
  • Use high fiber diet that produces high roughage.
  • Sitz bath to reduce pain and spasm.
  • Haemorrhoids that prolapse should be reposed gently and not forced back.
  • Take treatment for any disease that promotes straining at micturition like benign hypertrophy of prostate.

References

Benign Diseases of the Anorectum. In: Maingot’s Abdominal Operations. Zinner MJ, Schwatz SI, Ellis H (eds), 10th Edition, 1987, Prentice Hall International, pp 1437-1444.

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Fistula-In-Ano

Fistula in ano is a tract lined by granulation that connects superficially the skin around the anus and deeply the anal canal or the rectum. Low level fistula opens into the anal canal below the anorectal ring. The high level fistula opens into the canal at or above the anorectal ring. It is important to know the level of fistula since a low level fistula can be laid open without fear of incontinence

Salient features

  • Persistent seropurulent discharge that may be blood stained.
  • Pain and sometimes a history of a perianal abscess that has been drained.
  • Fistula in ano may be associated with tuberculosis, Crohn’s disease, carcinoma, bilharziasis.
  • There is usually an opening within 3-4 cm of the anal orifice with granulation tissue. The fistula heals only to recur later on. Digital examination may reveal the internal opening.

Treatment

Nonpharmacological

Local hygiene and sitz bath. Diet modification to avoid constipation.

Pharmacological

  1. Cap. Ampicillin 500 mg every 6 hours.
  2. Tab. Metronidazole 400 mg every 8 hours.
  3. Bulk laxative to relieve and avoid constipation.
    Definitive treatment is fistulotomy (laying open of the fistula tract), fistulectomy (excision of the fistula tract) and use of Seton. Secondary fistula needs treatment of primary disease. High level fistula may need proximal colostomy for treatment.

Patient education

  • Do not take treatment for anal disorders like abscess and fistula from unqualified persons.
  • Avoid constipation and take bulk laxatives.
  • Maintain local hygiene.

Reference

Benign Diseases of the Anorectum. In: Maingol’s Abdominal Operations, Zinner MJ, Schwatz SI, Ellis H (eds), 10th Edition, 1997, Prentice Hall International. London, pp. 1437-1454.

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Fissure-In-Ano

An anal fissure is an elongated ulcer in relation to anal canal. It most commonly occurs in the midline posteriorly. Most cases are idiopathic and may be due to trauma and ischaemia. Specific causes of fissure are incorrect operation for haemorrhoids, inflammatory bowel disease and sexually transmitted diseases. These can be acute and chronic.

Salient features

  • The severe pain on defecation that promotes constipation.
  • Bleeding is usually small and occurs as a streak by the side of stools.
  • A foul smelling discharge is present in chronic cases.
  • On examination, a longitudinal ulcer is seen in the midline posteriorly that may be covered by a skin tag. There is local inflammation and induration.

Treatment

The aim of the treatment is to obtain complete relaxation of the sphincter and provide relief from pain.

Nonpharmacological

  • Sitz bath- sitting in a tub containing luke warm water with potassium permanganate to provide relief from spasm and pain.
  • Local hygiene.
  • High fiber diet to prevent constipation.

Nonsurgical

  • 2% Glycerine trinitrate as an ointment for local application.
  • Manual dilatation of the anal sphincters-Lord’s procedure.

Surgical

  • Lateral anal sphincterotomy.
  • Dorsal fissurectomy and sphincterotomy.
    Complication of surgical treatment could include mild incontinence and prolonged healing time.

Patient education

  • Local care of the region and sitz bath should be regularly taken.
  • Avoid constipation by the use of high fiber diet and use of purgatives.

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Scrotal Swellings

Scrotal swellings can be either congenital or acquired. The acquired scrotal swellings could be further classified as inflammatory, traumatic or malignant. Important diagnoses include hydrocele, epididymo-orchitis, torsion of testis and tumours.

A. Hydrocele

This is a collection of fluid in some part of processus vaginalis usually tunica. It can occur in children and adults. Hydrocele could be primary or secondary to testicular diseases like inflammation, infections or malignancy. It can be unilateral or bilateral.

Salient features

  • Cystic swelling usually translucent, it is possible to reach above the swelling and it is not possible to feel the testis distinct from the swelling. Although there is history of reduction of size in children, it is not reducible.
  • Complications include rupture, haematocele formation, infection (pyocele), calcification and testicular atrophy and herniation through the dartos muscle in long standing cases.

Treatment

In infants, it is advised to wait till the age of two years to allow spontaneous resolution. Beyond the age of two years, the surgical treatment entails herniotomy by the inguinal approach. In adults, definitive treatment requires drainage of the fluid along with eversion of the sac with or without excision of the same. This can be done under local or regional anaesthesia.

B. Epididymo-Orchitis

Epididymo-orchitis is inflammation of the epididymis and the testis due to various causes. It can be acute or chronic. Infection reaches the epididymis via the vas from the lower urinary tract. A history of urinary tract infection is usually available. The condition has to be differentiated from torsion of testis (as given below).

Salient features

  • The epididymis and the testis show swelling with shiny edematous skin and tenderness. It may be possible to feel the epididymis and testis separately. The pain is relieved by rest and elevation of testis.
  • Urine examination shows pus cells. Complications include secondary hydrocele with clear fluid, abscess formation and pus discharge from sinus formation.

Treatment

Bed rest and scrotal support.

Pharmacological

  1. Cap. Doxycycline 100 mg once daily for 8- 10 days. It may be changed according to urine culture and sensitivity.
  2. Analgesic and antipyretics may be required.

C. Torsion Of Testis

Torsion of testis is most common between ages of 10- 25 years though it may occur at any age.

Salient features

  • There is sudden onset of pain in the affected testis and lower abdomen.
  • The testis is tender; lies higher as compared to its counterpart; the opposite testis lies horizontally; it isnot possible to palpate testis and epididymis separately; pain increases on elevation of testis and secondary haemorrhagic hydrocele.
  • Ultrasound examination and colour Doppler examination demonstrate torsion of testis and resultant obstruction of the blood supply.

Treatment (Immediately refer to a higher centre)

Treatment of torsion of testis requires immediate correction by surgical exploration through scrotal incision, untwisting of the cord and orchiopexy.

It is important to fix the opposite testis at the same time. It is of paramount importance not to delay the exploration even if diagnosis is doubtful or for the want of special investigations. Any undue delay can lead to gangrene of the testis.

Patient education

  • Any scrotal swelling should be brought to notice of your doctor.
  • Any sudden onset swelling of the testis merits immediate attention of the surgeon and delay in diagnosis or treatment even for few hours can be harmful.

References

  1. Disorders of the Testis, Scrotum & Spermatic Cord. In: Smiths General Urology, Tanagho EA, McAninch JW. (eds), 15th Edition, 2000, McGraw Hill Company Inc., New York, pp 6784-693.

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Inguinal Hernia

Hernia occur due to raised intra-abdominal pressure due to various causes or weakness of the body wall due to any disease. A hernia consists of the sac, the coverings and the contents of the sac that could be omentum, intestine, circumference of intestine, ovary or Meckel’s diverticulum. Most common type of the external hernia is the inguinal hernia, less common being femoral and umbilical. Therefore, management of inguinal hernia is discussed.

Salient features

  • Pain and swelling in the groin. The swelling increases as the duration of hernia increases.
  • Complications of hernia include irreducibility, incarceration and obstruction, strangulation and inflammation due to inflammation of the contents.

Treatment

Surgical treatment

The treatment of choice for hernia is surgical repair. The surgery is advocated as soon as the diagnosis is made since the complications are common. Even in children, hernia repair is done at the earliest after diagnosis. Any predisposing factors need to be treated first before hernia repair else recurrence is possible. The hernia with complications need to be operated in emergency.

Treatment in children entails herniotomy while in adults repair of the posterior wall of the inguinal canal without (herniorrhaphy) or with prosthesis (hernioplasty) after high ligation and division of the sac is done. This can be done by open repair or laparoscopic repair by the experts. Day care surgery under local anaesthesia is practiced at many centers.

Complications of herniorrhaphy include infection, haematoma formation, injury to viscera like urinary bladder, injury to vas and recurrence.

Nonsurgical treatment -Truss is not advocated for the treatment of hernia except in the extremely frail patients unfit for surgery or where surgery is refused by the patient. Application of external pressure causes trauma to skin and may cause injury to the contents.

Patient education

  • Reduce weight and quit smoking before surgery.
  • Treatment of any predisposing factors like chronic cough, prostatic enlargement and constipation is necessary.
  • The surgery should not be delayed since complications of hernia are frequent and can be serious.
  • After surgery, avoid lifting heavy weights, cycling etc. for three months.

References

Hernias. In: Maingot’s Abdominal Operations, Zinner MJ, Schwartz SI, Ellis H (eds), 10th Edition, 1997, Prentice Hall International, pp 497-580.

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Retention Of Urine

Retention of urine is inability to pass urine. It can be either acute or chronic.

  1. Mechanical causes of retention are: posterior urethral valves, foreign bodies, tumours, blood clot and stones, phimosis, paraphimosis, trauma (rupture of urethra or bladder), urethral stricture, urethritis, meatal ulcer, tumours, prostatic enlargement-benign or malignant, retroverted gravid uterus, fibroid, ovarian cyst, faecal impaction.
  2. Neurogenic- Postoperative retention, neurogenic bladder, spinal cord injuries, hysteria, drugs- anticholinergics, antihistaminics, smooth muscle relaxants.
Salient features

  • Acute retention of urine is characterized by inability to pass urine despite urge, suprapubic discomfort or severe agonizing pain. There may be previous such episodes or history of trauma, instrumentation or surgery.
  • Chronic retention is an enlarged painless bladder whether or not the patient is having difficulty with micturition. Some times acute episode can be precipitated in cases of chronic retention of urine.
  • There may be symptoms suggestive of prostatic enlargement in elderly male.
  • On examination, there is suprapubic swelling arising out of pelvis in the midline in the hypogastric region that is dull to percussion and cystic in nature. This helps to differentiate from anuria where urinary bladder is not palpable.
  • Rectal examination will help to confirm the prostatic pathology in elderly patients. Spinal defects or neurological findings suggest presence of neurogenic bladder.

Treatment

  1. General measures include sedation, adequate hydration and antibiotics if sepsis is present.
  2. If there is history of trauma, urethral injury should be ruled out before attempting catheterization.
  3. If the urethra is patent, a catheter is passed in to the bladder under strict aseptic precautions and is connected to a sterile closed collecting system. The catheter is chosen according to the size of the external meatus. In cases of acute retention, single catheterization is adequate or an indwelling self-retaining catheter is inserted if deemed necessary.
  4. If urethral pathology is present or there is inability to pass the catheter, a suprapubic puncture or cystostomy is performed to relieve the retention.
  5. In case of chronic retention, decompression should be performed intermittently (300-400 ml volume) to avoid haematuria that can occur after sudden decompression.
  6. The patient should be kept under observation after admission for investigation to elucidate the cause of retention. The investigations include urine examination, renal functions, plain and contrast radiological studies, ultrasound, CT scan or MRI. Urodynamic studies are required to diagnose neurogenic bladder. Cystoscopy can help to diagnose and treat many conditions of the urethra and urinary bladder.
  7. Definitive treatment of the aetiology is done after proper investigations.

Pharmacological

  1. Tab. Cotrimoxazole (960 mg) 2 times a day
    Or
    Tab. Norfloxacin 400 mg 2 times a day for 5-7 days. This may be changed according to urine culture and sensitivity reports.

Patient education

  • Explain catheter care-measures – tip of the urethra should be cleaned with antiseptic solution regularly.
  • Watch for blood in urine.

References

  1. The Urinary bladder. In: Bailey & Loves Short Practice of Surgery, Russel RCG, Williams NS, Bulstrode CJK. Arnold (eds), 23rd Edition, 2001, pp 1204-1208.

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Appendicitis

Appendicitis is the commonest cause of acute abdomen and may appear as catarrhal appendicitis or as obstructive appendicitis and sometimes it may present as an appendicular lump or appendicular abscess or as burst appendix with peritonitis.

Salient features

  • Acute central abdominal pain, followed by nausea, vomiting & fever, with the pain after a variable period, shifting to right lower abdomen localized tenderness maximum at the Mc Burney’s point, rebound tenderness and guarding in the right iliac fossa.
  • An inflammatory lump in the right lower abdomen or signs of peritonitis.
  • A polymorphonuclear leucocytosis and ultrasonographic appearances may help to corroborate the clinical diagnosis.
  • Investigations are primarily undertaken to exclude other conditions like ectopic gestation or ureteric calculus.

Treatment

The definitive treatment is appendicectomy & the sooner it is done, the better. The surgery should be delayed if the patient is moribund with advance peritonitis where the conservative measures will need to be supplemented by measures to make him fit for operation. An interval appendicectomy should be performed where a lump has formed or when attack has already resolved or circumstances make surgery not feasible.

Nonpharmacological

Stop oral feeding.

Pharmacological (Expectant management)

  1. Intravenous fluids to maintain hydration. Requirement of fluids would be more if the patient has peritonitis & septicaemia.
  2. Inj. Ciprofloxacin infusion (100 mg/50 mg) 100 ml twice a day for 5 days.
  3. Inj. Gentamicin (40 mg/ml), 80 mg IV 8 hourly.
    Or
    Inj. Amikacin (500 mg/2 ml), 2 ml IV twice a day.
  4. Inj. Metronidazole infusion (500 mg/100 ml) 100 ml IV 8 hourly.
  5. Inj. Diclofenac sodium (25 mg/ml) 50 ml IM SOS.
    (CAUTION: Purgation & enema are contraindicated)
    Pain subsides first, followed by relaxation of the abdomen and control of fever. Tenderness disappears later. Polymorphonuclear leucocytosis tends to settle down. Failure of signs & symptoms to subside or the appearance of new signs & symptoms during expectant treatment, calls for surgical intervention

Postoperative Management

  • Oral feeding is started when abdomen is soft, the patient has passed flatus/stools and bowel sounds have appeared. Start with liquids, gradually permitting semi solid & solid diet over a period of 2-3 days.
  • Antibiotics should continue for 5 days or more if the condition demands.
    Initially they are given by parenteral route and later switched to oral route when the patient starts tolerating semi solid diet.
  • Patient is discharged usually between 3rd & 5th postoperative day, when he is comfortable, ambulatory, tolerating semi solid or solid food, afebrile and has a healthy wound.
  • Sutures are removed around 7th postoperative day.

Patient education

  • Normal routine physical work can be permitted in 10-15 days (5-7 days after laparoscopic appendicectomy).
  • Moderate physical work is permitted after 4-6 weeks (2 weeks after laparoscopic appendicectomy).
  • Heavy physical work is permitted after 2-3 months (4-6 weeks after laparoscopic appendicectomy).

References

Appendix and Appendicectomy. In: Maingot’s Abdominal Operations, Zinner MJ, Schwartz SI, Ellis H (eds), 10th Edition, 1977, Prentice Hall International. pp 1191-228.

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Cholelithiasis

Most patients with cholelithiasis have stones exclusively in the gall bladder, but 15% of patients, in addition, have common bile duct (CBD) stones.

Salient features

  • Most patients are asymptomatic, some patients present with dyspepsia or vague epigastric pain. The remaining patients may present with recurrent biliary colics or with acute cholecystitis or with symptoms of CBD disease or gall stone or pancreatitis.
  • Obesity, fertility and diabetes are known to increase the chances of gall stone formation.
  • Diagnosis made clinically and confirmed by ultrasonography.

Treatment

Low fat diet with no spices.
Definitive treatment is cholecystectomy in symptomatic and asymptomatic patients with diabetes or a solitary large stone or multiple small stones with wide cystic duct or porcelain gall bladder or anxious patients. If the patient comes after 48 hours manage conservatively and cholecystectomy after 6-8 weeks.
Expectant management – In case of acute cholecystitis, empyema gall bladder, and stones in the CBD.
Maintenance IV fluids (for details see section on fluid and electrolyte imbalance)

  1. Inj. Ciprofloxacin (Infusion 100 mg/50 ml) 100 ml IV twice a day.
  2. Inj. Gentamicin (40 mg/ml) 2 ml IV 8 hourly.
    Or
    1. Inj. Ampicillin (500 mg/ml) 1 ml IV 6 hourly.
    2. Inj. Cloxacillin (500 mg/ml) 1 ml IV 6 hourly.
    Or
    1. Inj. Ciprofloxacin (Infusion (100 mg/50 ml) 100 ml IV twice a day.
    2. Inj. Amikacin (500 mg/2 ml) 2 ml twice a day.
  3. In case anaerobic bacterial infection is suspected or anticipated, give Inj. Metronidazole (500 mg/100 ml) 100 ml IV 8 hourly.
  4. Inj. Diclofenac sodium (25 mg/ml) 2 -3 ml IM SOS or 6 hourly.
    Or
    Inj. Pentazocine lactate (30 mg/ml) 1 ml IM SOS.
  5. Inj. Hyoscine butylbromide (20 mg/ml) 1 ml IV SOS.
  6. In patients having obstructive jaundice, add Inj. Vitamin K (10 mg/ml) 1 ml IM once or twice a day till prothrombin time reaches to a satisfactory level.
    Antibiotics are usually stopped after 5-7 days unless the patient has evidence of persistent infection or has indwelling tube (e.g., T-tube).

Patient education

  • To avoid fatty & fried meals for 3 months.
  • Although ambulation is encouraged as early as possible, heavy physical exertion should be avoided for 2 weeks (after laparoscopic cholecystectomy) and for 3 months after conventional cholecystectomy.
  • If T-tube has been placed, it should be removed after 2-3 weeks, after ensuring that the CBD is patent, non dilated and there is free flow of contrast into the duodenum.

References

Cholelithiasis and Cholecystectomy. In: Maingot’s Abdominal Operations, Zinner MJ, Schwartz SI, Ellis H (eds), 10th Edition, 1977, Prentice Hall International, pp 1717-1738.

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Acute Abdomen

Abdominal pain can occur due to variety of medical and surgical causes.

It is important to elicit a detailed clinical history and perform abdominal examination to determine the cause of pain. In very severe cases, it may be necessary to give treatment before proper history can be obtained or examination is allowed by the patient.

Causes of acute abdomen

Abdominal causes

  1. Inflammation of peritoneum due to bacterial or chemical contamination. Perforation of appendix or bowel, ulcer, pancreatitis or pelvic inflammatory disease.
  2. Mechanical obstruction of hollow viscera-intestinal obstruction, ureteric obstruction due to stone or other causes, obstruction of the biliary tree.
  3. Vascular disturbances – vascular rupture, embolism or thrombosis, torsion of pedicle.
  4. Abdominal wall injury or infection in the muscles. Referred pain in case of pneumonia, angina, spine-radiculitis and torsion of testis.

Metabolic causes

Insect bite, lead poisoning, uremia, diabetic ketoacidosis, porphyria and other allergic causes.

Neurogenic causes

  1. Organic- Tabes dorsalis, Herpes zoster, neuralgia.
  2. Functional.

Approach to the patient with acute abdomen

  1. In view of the variety of causes, it is absolutely essential to elicit an orderly detailed history about the nature of pain and perform a sequential examination. Narcotics or analgesics should be withheld till the diagnosis is made.
  2. A proper history can provide more valuable information than the investigations. The features of the history that must be elicited are site, time and nature of onset, severity, nature of pain, progression, duration, relieving factors, exacerbating factors and radiation. History of GI complaints like vomiting, constipation, haematemesis and melena and urinary complaints should be obtained.
  3. Examination of the patient with respect to facies, posture in the bed, respiration can help in localizing the cause and nature of pain. The abdominal examination should be thorough but extremely gentle starting from the nontender area. Points to be noted are area of maximum tenderness, guarding, rebound tenderness, bowel sounds, obliteration of liver dullness and presence of any associated mass or free fluid in the abdomen.
  4. It is important not to miss examination of supraclavicular lymphnodes, hernial orifices, peripheral pulsations, genitalia, bowel sounds and rectal and/or vaginal examination.
  5. Laboratory investigations depend upon the suspected cause of pain and include haemogram, kidney function tests, liver function tests, serum or urinary amylase and lipase, blood sugar and urinalysis. Special analysis like serum lead estimations may be needed if deemed necessary.
  6. Radiological investigations include plain radiographs of abdomensupine and erect and chest. In a sick patient, lateral decubitus X-ray may provide the same information. Ultrasound, computed tomography and special radiological investigations may be done as and when required.
  7. Invasive procedures like endoscopy and laparoscopy are done only if indicated.
  8. It may be necessary to rush the patient to operation room if there is suggestion of intra-abdominal haemorrhage along with pain. Any delay for history, examination and investigations can cause more harm to the patient.
  9. There may be occasions when the cause of the pain cannot be determined on the basis of clinical examination and investigations. Under such circumstances, it is prudent to provide pain relief to the patient and keep him under observation to watch for development of new signs or symptoms.

Treatment

Treatment of acute abdomen depends upon the cause of pain. In case of medical causes of pain, the offending cause is removed or treated appropriately.

Pharmacological

This depends upon the aetiology of pain.

  • In soft tissue lesions or trauma NSAIDs are prescribed.
  • In obstruction or colicky pain due to spasm, Tab. Dicyclomine SOS.
  • Initially, injectable administration may be needed for severe pain and later it may be switched to oral route.
  • Narcotics may be given in case of severe pain.
  • Antibiotics are given for inflammatory causes of pain.

Surgical

Any acute surgical cause of acute abdomen like perforation peritonitis, appendicitis, perforation of ulcer etc. should be treated immediately and appropriately.

Patient education

  1. Do not administer analgesics for acute abdomen on your own. This may delay treatment of surgical cause.
  2. Enema should not be self-administered for constipation without advise of the treating physician/surgeon.
  3. Narcotic analgesics should be taken for minimum period as long term intake may lead to addiction.

References

Abdominal Pain. In: Harrison’s Principles of Internal Medicine, Braunwald E., Fauci AS, Kasper DL et al (eds), 15th Edition, 2000, McGraw Hill Company Inc., New York, pp 67-70.

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Dysphagia

Dysphagia is the sensation of difficulty in swallowing. It may be due to general causes e.g. myasthenia gravis, bulbar palsy, hysteria etc.) or due to the local causes. The latter may be

  1. Intraluminal (e.g. foreign body)
  2. Intramural (e.g. achalasia, oesophagitis, oesophageal strictures, Plummer-Vinson syndrome, pharyngeal pouch, benign neoplasm, malignant neoplasm) or
  3. Extra luminal (e.g. retrosternal goitre, mediastinal tumor, mediastinal lymphadenopathy, aortic aneurysm, hiatus hernia).
Salient features

  • Difficulty in swallowing (solids and/or liquids), oesophageal pain, regurgitation and aspiration.

Investigations

  • Barium swallow to evaluate cause, site & extent of the lesion and the state of the oesophagus above & below the lesion; upper GI endoscopy for direct vision evaluation and for taking tissue for histopathological examination wherever indicated.
  • Abdominal ultrasound, chest x- ray and other routine investigations.
  • CT scan and endoscopy ultrasound to be considered in tertiary care centres, wherever indicated; and oesophageal manometry, pH studies and evaluation for H. pylori to be considered in tertiary care centres, wherever indicated.

Treatment

Definitive treatment depends on the cause and its extent.

Nonpharmacological

  • Diet restricted to liquids or semi solids depending upon extent of dysphagia.
  • Psychotherapy if the patient is depressed or demoralized.

Pharmacological

Gel Magnesium hydroxide + Aluminium hydroxide + Activated
Dimethicone (250 mg + 250 mg + 50 mg/ml) 20 ml 6 hourly.
Or
Tab. Ranitidine 150 mg 2 times a day.

References

Dysphagia. In: Harrison’s Principles of Internal Medicine, Braunwald E, Fauci AS, Kasper DL, Hausen SL et al (eds), Vol. 1, 15th Edition, 2001, McGraw Hill Company Inc., New York, pp 233-235.

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Breast Abscess

Breast abscesses can be classified into mastitis neonatorum, lactating epidemic or sporadic mastitis, and non-lactating breast abscesses.
Usually caused by highly virulent strains of penicillin resistant Staphylococcus aureus and anaerobic Streptococci.

Treatment

Nonpharmacological

Rest and support to the breast and to continue breast feeding from both the breasts, however, in case of larger abscess shift to bottle feeding.

Pharmacological

In early stage (induration only):

  1. Tab. Erythromycin 500 mg 3 times a day for 7 days.
  2. Tab. Metronidazole 400 mg 3 times a day for 7 days.
  3. Tab. Ibuprofen 400 mg as and when required.

In case of no improvement or large abscess:

  1. Antibiotics as above.
  2. Incision and drainage of pus through thinned skin over the abscess (Large abscesses require operation under intercostal block or general anaesthesia).
  3. Daily dressing.
    In some cases suppress lactation with hormones if the mother finds
    breast feeding too painful.

Patient education

  • To maintain good hygiene and to continue breast feeding from both the sides unless it is a large abscess and very painful.
  • Advise on timely weaning of the infant.

References

Abscesses in Special Sites. In: Hamilton Bailey’s Emergency Surgery, Ellis BW, Paterson-Brown S. Arnold (eds), 13th Edition, 2000, London pp 165-166.

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Thyroid Swelling

Thyroid swelling forms one of the most important differential for swelling in front of the neck. The differential diagnosis of thyroid swelling are benign goiter, intrathyroid cysts, thyroiditis, benign and malignant tumours. Simple goitre is enlarged thyroid gland and occurs commonly around puberty in girls due to iodine deficiency. Malignancy should be suspected in case of extreme of age, male sex, rapidly growing swelling, persisting pain, dysphagia, recurrent laryngeal nerve palsy, hardness and fixity of the thyroid gland and presence of one or more palpable neck nodes. Fine needle aspiration cytology, isotope scan and ultrasonography are helpful in differentiating the causes of thyroid swelling.

Treatment

Simple diffuse hyperplastic goitre is preventable by using iodised salt.

Treatment with L-thyroxine can reverse the swelling at the stage. Simple nodular goitre is treated by subtotal thyroidectomy (for details see Figure 1).

Figure1. Management plan for thyroid nodule



Thyroidectomy

Preoperative care

Preoperative antibiotic prophylaxis (Inj. Ampicillin 1 g IV 30 min before operation) is given to the patient. Preoperative thyroidectomy indirect laryngoscopy (IDL) is performed to identify compensated or unsuspected recurrent laryngeal nerve palsy. Before operation, thyrotoxic patient should be made euthyroid with antithyroid drugs (carbimazole 0-15 mg 4 times a day and propranolol 20 mg 3 times a day). Fully discuss the potential complications with the patients – mentioning the risk to parathyroid gland and recurrent laryngeal nerve.

Postoperative care

Place patient in a slightly propped up position. Carefully observe for respiratory insufficiency, haemorrhage from the wound, irritability to the facial nerve and carpopedal spasm (parathyroid injury). Monitor drain output daily and remove if 24 hours output becomes lesser than 10 ml. Check wound site for infection and suture removed on the 5th day.

Complications

The most immediate life threatening complication is haemorrhage under deep cervical fascia, which can lead to acute asphyxia. Management include reopening of the suture line, drain the haematoma and reexploration for control of bleeders. Damage to recurrent laryngeal nerve can lead to respiratory distress (bilateral recurrent laryngeal nerve) and hoarseness of voice. Parathyroid damage leads to hypocalcaemia.
Symptomatic hypocalcaemia (positive Chovstek’s or Trousseau’s sign) or corrected serum calcium level < 8 g/dl is treated with 10% calcium gluconate intravenously. If hypocalcaemia persist, oral calcium supplement and synthetic Vitamin D is necessary.
Late complication includes recurrent thyrotoxicosis (Grave’s disease), hypothyroidism, recurrence of malignancy at the local site or in the lymph nodes in the neck.

Radioiodine therapy is indicated in follicular, papillary and mixed carcinoma. Following total thyroidectomy, a total body radioactive isotope scan should be arranged four weeks after the operation. During this period L-thyroxine therapy should be withheld. If radioactive scan shows residual thyroid tissue or metastatic deposit then further dose of radioiodine should be given to ablate these. Following isotope scan, high dose L-thyroxin (0.2-0.3 mg) should be started and continued for life. Radioactive iodine has no role in residual/metastatic medullary carcinoma. Treatment approach to Hurthle cell neoplasm is similar to follicular neoplasm.

Follow up

Patients should be followed at three monthly intervals for the initial 2 years and 6 monthly for next three years and then at yearly interval for life. On each follow up visit patient should be examined for any local or nodal recurrence in the neck, a chest X-ray should be done to exclude pulmonary deposit and clinical features of thyroid toxicity noted and dose of L-thyroxin regulated.

References

1. The Thyroid Gland. In: The New Aid’s Companion in Surgical Studies. Burnand KG, Young AE (eds), 2nd Edition, 1998, Churchill Livingstone, London pp 459-485.

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Cervical Lymphadenopathy

An enlarged cervical lymph node is the commonest cause of lump in the neck. Cervical lymph nodes may become enlarged as a result of inflammation or neoplastic process (Table 1). Tuberculosis is the commonest cause of cervical lymphadenopathy.

Table 1. Causes of lymphadenopathy and clinical features

Condition Causes Clinical features
Acute inflammation Infection of the aerodigestive tract, head and neck or other infections. Fever, sore throat, firm,tender nodes 1-2 cm in diameter
Chronic inflammation Tuberculosis, sarcoidosis, Histiocytosis X Swelling in the neck and fever, cough may or may not be present
Variable on presentation
depending on the stage of the disease: multiple matted lymph nodes/
cold abscess
Lymphomas Hodgkins/Non Hodgkins lymphoma Large painless rubbery lymph nodes.
Metastatic Carcinomas of the upper aerodigestive tract, skin tumours of the head and neck:squamous cell carcinoma, basal cell carcinoma, melanoma. Symptoms related to primary disease, firm to hard lymph node enlargement

Treatment

Detailed history and examination are essential to pinpoint specific aetiology. Majority of the lymph nodes are reactive to viral infections of upper respiratory tract, therefore, do not require any treatment.

  1. In case of acute suppurative lymphadenopathy secondary to any bacterial focus of infection in the drainage area.
    Cap. Cephalexin 250-500 mg every 6 hours for 7 days.
    Or
    Cap. Amoxicillin 250-500 mg every 8 hours for 7 days.
    If lymph nodes persists, perform fine needle aspiration cytology (FNAC) and treat accordingly. If FNAC is nonconclusive take a biopsy from the enlarged lymph
    node and treat accordingly.
  2. In case of chronic lymphadenopathy perform FNAC and treat accordingly. If FNAC is nonconclusive, perform biopsy and treat accordingly.

Treatment (Tubercular lymphadenopathy)

Start antitubercular therapy (see section on tuberculosis(tuberculosis, cutaneous,meningitis,other)) and reassess the patient after 6 months. If lymph nodes are either not present or less than 1 cm size keep the patient under follow-up and continue treatment.

However, if lymph nodes are palpable and more than 1 cm take a biopsy of the node and treat accordingly and consider second line antitubercular drugs.

References

The Neck. In: The New Aid’s Companion in Surgical Studies, Burnand KG, Young AE, 2nd Edition, 1998, Churchill Livingstone, London pp 451-455.

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Varicose Veins

Chronic venous disease of the lower limb is one of the most common conditions affecting the adults. Varicose veins the most common complaint, represent one end of the spectrum of venous disease which extends through increasing degrees of venous insufficiency and may result in leg ulceration in the most severe cases.

Basle Study Venous Classification

No venous disease.
Varicosities

  1. Telangiectasia/hyphenwebs (spider veins): intradermal varicose veins that are small and rarely symtomatic.
  2. Reticular veins: subcutaneous veins that begin at the tributaries of the trunk veins.
  3. Trunk veins: varicose veins of the greater/lesser saphenous system and its named tributaries.

Chronic venous insufficiency

  1. Dilated subcutaneous veins.
  2. Hyperpigmented/depigmented areas.
  3. Open/healed ulcer.
Salient features

  • Varicose veins refers to any dilated, tortuous, elongated vein, regardless of size.
  • Duplex ultrasonography is confirmatory and also helps in
    localization of perforators and at times saphenopopliteal opening which is of immense help in the performance of operation.
    Evaluation of the deep venous system is a must in a patient with a history or clinical examination suggestive of deep venous thrombosis (leg edema, present or past ulcer).

Treatment

Carefully examine the lower limb for sapheno-femoral reflux, varices and perforators in thigh and manifestations of venous insufficiency in calf and foot. Reassurance, the use of elastic compression stockings, injection sclerotherapy or surgical treatment.

Surgical

A recent episode of deep venous thrombosis is a contraindication for operation in the superficial venous system. However, in patients with old deep vein thrombosis (DVT), perforator or obviously varicose long or short saphenous system should be ligated or treated by sclerotherapy. The most definitive approach in the thigh perforators is flush ligation and thigh stripping, with careful attention to groin tributaries. Saphenofemoral ligation alone can be performed under local anaesthesia but the addition of stripping or operation of the sapheno popliteal system warrants spinal/general anaesthesia.

In case of small varices and those where the main long and short saphenous veins and their major tributaries, are competent, injection sclerotherapy with STD (sodium tetradecyl sulphate) is best used in the management of large varicose veins and perforators in the calf. Treatment can be repeated when necessary.

Technique: Place 25G needle into the varices or perforators. Empty the vein and inject 0.5 ml of sclerosant i.e. STD. Compression is applied immediately with compression bandage or stocking.
Or
Ambulatory phlebectomy (avulsion of veins) prevents venous recanalization with recurrence.

Postoperative management

  • Compression bandaging immediately following stripping or avulsion of veins. Replace bandages by compression stocking after 2 days.
  • Limb elevation and encourage the patient to walk with compression stockings after first change of dressing 48 hours after operation.
  • Postoperative pain is controlled with dextropropoxyphene or NSAIDs.

Patient education

  • Certain do’s are leg exercise, leg elevation, wear stockings and drinking 4-5 L of fluids in a day.
  • Certain don’ts are hot bath, exposure to extreme of temperatures, pregnancy, contraceptive pills and estrogens, long journeys (flight).
  • Teach the patient leg exercises-frequent movements of toes and heels, Sarvangasan or Shirshasan, and elevation of foot end of the bed about 6 inches by putting a block of wood or 2 bricks under foot end of bed.
    To avoid prolonged standing or dangling legs down.

References

Venous Disorders. In: Bailey and Love’s Short Practice of Surgery, Russel RCG, Williams NS, Bulstrode CJK. Arnold (eds), 23rd Edition, 20000 London, pp 235-255.

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Wound Care

Wounds can be classified as acute or chronic. Acute wounds heal uneventfully within an expected time frame e.g. burns. For the purpose of guidelines, chronic wounds can be defined as an ulcer present at least for 6 weeks.

Treatment

Acute wounds

The overall objectives in caring for wounds that are incompletely clotted are to minimize unnecessary blood loss and to avoid the formation of a haematoma.

  1. Irrigate gently with copious quantities of water or normal saline. Debris and necrotic tissue should be removed without damaging healthy tissue.
  2. Healing of acute wound is further facilitated by closure. Alternatively, closure can be delayed for several days to allow infection to clear.

Chronic wounds

Identify and treat the predisposing factors e.g. diabetes mellitus, peripheral arterial or venous disease, severe anaemia, protein deficiency, rheumatoid arthritis, systemic vasculitis, Cushing’s syndrome and conditions requiring systemic steroid therapy

Type of ulcer Clinical assessment Investigation
Arterial Involves deep fascia or deeper Doppler
structure, decreased or absent distal pulses US(ABPI<0.8)
Venous Involves skin, subcutaneous fat Tortuous long/short saphenous veins Perforators incompetence Doppler US/Venography
Tubercular Undermined edge Edge biopsy
Trophic Punched out/undermined
Malignant Raised/everted margin Edge biopsy
No evidence of granulation tissue

Nonpharmacological

  • Encourage daily or twice a day bath, to avoid walking bare foot or with slippers and patient should be encouraged to wear shoes and socks.
  • Patients with leg ulcer to reduce standing or excessive walking. In leg ulcer due to chronic venous insufficiency or edema, patients should be advised to wear elastic stockings, elevation of leg and foot end of the bed while asleep along with some leg exercises to activate the calf muscle pump.

Pharmacological

Identify the microorganism and treat accordingly. Tubercular ulcer is treated with antitubercular drug (2HRZE+7HR) for at least 9 months.

Surgical treatment

Surgical debridement in ulcers associated with necrotic tissue or slough. Clean the wound with physiological normal saline or tap water only (antiseptics delay wound healing).

Daily dressing
Gauze adheres to the wound bed and it may remove viable tissue from the wound surface on removal, resulting in delayed wound healing.
Occlusive (moisture retentive) dressings (Hydrocolloid gel) in case of clean and shallow ulcers without any pus discharge or other features of infection. Occlusive dressings have barrier properties that enable to prolong the presence of moisture and wound fluid in the wound bed.
Calcium alginate dressing: For bleeding wounds and wounds with a cavity.
Refer patients with chronic leg ulcer to a vascular surgeons general surgeons or with some experience in peripheral vascular problems for surgical treatment.

Patient education

  • To prevent ulcers in future, explain about the care of leg ulcers, wearing socks, shoes and compulsion stocking.
  • Good personal hygiene (daily bath) and after the bath the healed scar area should be massaged with an emollient cream such as lanolin or some other oil to keep the scar tissue soft and supple and prevent further breakdown.
  • Regular use of calf muscle activating exercises and leg elevation and to avoid prolonged period of standing or sitting with legs down.
  • In case of diabetic patients, control of diabetes is necessary.

References

Incisions, Closures & Management of Wound. In: Maingot’s Abdominal Operators. 10th Edition, 1987, Prentice Hall International, pp 395-426.

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Post Operative Care

Post operative wound infection

Wound infections are classified as:
Minor: e.g. stitch abscess, cellulitis.
Major: e.g. presence of discrete collection of pus in wound.
Superficial infections, are, limited to skin and subcutaneous tissue. Deep infections involve the areas of wound below the fascia.

  • Preoperative predisposing factors:
    Age, obesity, malnutrition, diabetes and malignancy.
  • Operative predisposing factors:
    Duration of surgery, type of operative wound.
    Clean – 1%, clean contaminated – 1-5%, contaminated 5-30%, dirty – more then 30%, poor surgical technique, break of sterilization.
  • Postoperative – cross infection in wards.
    Staphylococcus aureus is the most frequently involved organism. Other less common organisms are Enterococci, Pseudomonas, Proteus, E. Coli and Klebsiella.
Salient features

  • Pain is unusually severe for the magnitude of procedure and last long.
  • Fever: 101 to 102° F with tachycardia is usually present.
  • Local Examination: Wound is warm to touch and may be swollen and edematous. Redness of the surrounding area and cellulites is often present.
  • Wound infections are generally evident between 3rd to 6th postoperative day.

Treatment

Superficial Infections:

  1. Drainage: Wounds are managed by opening up the incision to provide adequate drainage.
  2. Dressing: Daily dressing with Povidone iodide 5% and a wick is placed to prevent pre mature closure of the wound.
  3. Analgesics: Tab. Ibuprofen 400 mg 3 times a day till pain is there.
  4. Deep Infections: antibiotics are given on the basis of pus culture and sensitivity in addition to drainage of wound.

Prevention

Postoperative wound infection rate can be minimized by adequate skin preparation, bowel preparation, prophylactic antibiotics, meticulous surgical technique

Postoperative pain relief

Postoperative pain is associated with all surgical procedures. This varies according to the surgical procedure. Severe pain can prolong gastrointestinal ileus, urinary retention, impair respiratory movements producing atelectasis and predisposes to deep vein thrombosis due to immobilization.

Various methods to alleviate postoperative pain are NSAIDs, opioids (intramuscular, transdermal or transmucosal), patient controlled analgesia, local infiltration of anaesthetic drugs, epidural analgesia and intrapleural analgesia. The method used depends upon the site and the magnitude of surgery done, severity of pain, whether the patient is allowed orally and facilities and expertise available. It is necessary to give analgesics by intramuscular or intravenous route in the immediate postoperative period and till the patient is able to accept orally.

Commonly used agents are:
Inj. Diclofenac sodium 75 mg 6-8 hourly.
Or
Inj. Pentazocine (30 mg/ml) 30-60 mg IM/IV repeated 3-4 hourly.
Or
Inj. Tramadol (50 mg/ml) IM/IV 4-6 hourly.
Or
Inj. Morphine (15 mg/ml) 10-15 mg, can be repeated 4-6 times.
In tertiary care centers, epidural analgesia , intravenous patient controlled analgesia, intrapleural analgesia can be used under expert care.
When patient is able to accept orally.
Tab. Paracetamol 500 mg 3-4 times a day.
Or
Tab. Ibuprofen 400-600 mg 8 hourly.
Or
Tab. Nimesulide 100 mg twice daily.

Postoperative nausea and vomiting

Postoperative nausea and vomiting lead to significant morbidity and prolonged hospitalization. It has an incidence of 20-30% after abdominal surgery. Predisposing factors are diabetes mellitus, pregnancy, dehydration, electrolyte imbalance, gastroesophageal reflux, emergency surgery, use of certain anaesthetic drugs and opioids.

Treatment

Bowel obstruction (mechanical or paralytic ileus) should be ruled out as a cause of vomiting by proper examination and investigations if it is associated with abdominal distension, fever and occur beyond 3rd postoperative day.

Nausea and vomiting are managed with bed rest, Intravenous fluids, analgesics to relieve postoperative pain, nasogastric decompression.

Pharmacological

Inj. Metoclopramide (5 mg/ml) 10 mg IM/IV 1-3 times daily or SOS.
Or
Inj. Ondansetron (2 mg/ml) 4 mg IV.
In children 0.15 mg/kg.
Or
Inj. Promethazine (25 mg/ml) 2 ml IV SOS.

Postoperative pneumonia

Pulmonary disorders remain the most frequent post operative problem and 10-15% of patients are considered to have clinically significant chest complication after surgery under general anaesthesia.

Factors predisposing to increased chest complications are smoking, obesity, chronic restrictive and obstructive lung disease, prolonged general anaesthesia and presence of nasogastric tube.

Postoperative pneumonia is caused by pathogens such as Pseudomonas, Serratia, Klebsiella, Proteus and Streptococcus.

Salient features

  • Fever, productive cough, dyspnoea, chest pain.
  • Bronchial breathing and presence of rales.
  • Chest X-ray shows areas of consolidation.

Treatment

  1. Antibiotics: depending upon sputum culture and sensitivity. Initial treatment can be started with aminoglycoside and antipseudomonas Cehphalosporins.
  2. Inj. Ketorolac 30 mg every 6-8 hours IV or IM
    Or
    Inj. Diclofenac 75 mg IM every 6-8 hours.
  3. Chest physiotherapy
  4. Nebulized bronchodilators: may be used if bronchospasm is present.

References

Preoperative and Postoperative Management. In: Maingot’s Abdominal Operation , Zinner MJ, Schwartz SI, Ellis H, 10th Edition, 1997, Prentice Hall International, pp 461-478.

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